I have a female that developed a spot on her muzzle that looks like a cigarette burn (black and scabbed up). The spot went away and then a bigger spot showed up on the underside of her ear. This one is about the size of a penny. Now other spots near the larger one are comming up and the one on her muzzle has reappeard as well.
These will scab up and then the scabs will break up and the "hole" gets deeper everytime.
Any Ideas?????
BTW....I don't know if it makes a difference but I switched feed for two bags until I could get to the place I normally buy my Joy.
Spots on one ear and muzzle
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Bunny Runner
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Have you noticed any scratching? If so, it sounds like you may have a case of Mange starting or it could be a bacterial infection of some sort. You really should take this dog to the vet to get it diagnosed. Mange typically starts on or around the face, ears, neck, underbelly, and front legs. The lesions will appear and then become heavily crusted and be accompanied by intense itching.
With the lesions becoming bigger each time the crust falls off, it could indicate a bacterial infection, which could be secondary, but either way, this dog should be see by a vet so treatment can begin for the infection (or whatever it is).
With the lesions becoming bigger each time the crust falls off, it could indicate a bacterial infection, which could be secondary, but either way, this dog should be see by a vet so treatment can begin for the infection (or whatever it is).
Education, Preservation, and Conservation ensures a "WILD" future for our children!
I coated her ear and muzzle with Blu Kote for bacterial infection.....she has been kenneled with other dogs since I got her and the first time I noticed it was when she got a cut on her ear and muzzle all in the same day. None of the other dogs have anything on them and she is not itching at all. It seemed to get better and now it has gotten worse....... 
If it does not clear up or start looking better in a week or so of using the Blu Kote I will be taking her to the vet.
BTW - she is a real light "blue/white" color and she seems to not heal well with any scratch or cut..... Is this normal?? I'm thinking the cuts have gotten infected or something.
If it does not clear up or start looking better in a week or so of using the Blu Kote I will be taking her to the vet.
BTW - she is a real light "blue/white" color and she seems to not heal well with any scratch or cut..... Is this normal?? I'm thinking the cuts have gotten infected or something.
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samlyn0001
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Seeings how you got yourself a "blue" pup you should check out this article.
Color Dilution Alopecia
By Teri Dickinson, DVM
Alopecia (hair loss) related to dilute coat color is a recognized condition in dogs. The currently accepted medical terminology for this condition is Color Dilution Alopecia (CDA). The condition may affect any dilutely pigmented dog, regardless of coat color. This condition was previously known as Blue Balding Syndrome, Blue Doberman Syndrome, Color Mutant Alopecia, Congenital Alopecia, etc. The term Color Mutant Alopecia arose because dilutes were at one time mutations from the deep pigment occurring in wild canines. Dilutes are now a regularly occurring form of pigmentation in many breeds and have been for hundreds of years. The term mutation is therefore not applicable to dilute individuals. References to Doberman Pinschers or blue hair coats arose because the condition is common in blue individuals of this breed, but it is not limited to either blue dogs or Dobermans. The term congenital means present at birth, but CDA affected dogs are born with normal hair coats.
The dilute (also known as Maltese) gene also appears in both mice and cats, and interestingly enough, is not associated with any abnormal coat conditions in those species.(1) Color Dilution Alopecia (CDA) has been recognized in dilute individuals of many breeds of dogs including Chow Chows, Dachshunds, Doberman Pinschers, Great Danes, Irish Setters, Italian Greyhounds, Standard Poodles, Salukis, Whippets, and Yorkshire Terriers.((2),(3),(4),(5)) Dilute individuals carry a recessive genotype of dd and are characterized by blue, bluish-grey, lavender or flesh-colored noses, lips and eye rims. The coat colors may include blue, fawn, blue-fawn, bronze, taupe or some variation of these. These dogs are usually easily distinguished from their deeply (non-dilute) pigmented counterparts. Deeply pigmented individuals carry a dominant genotype of Dd or DD and have black or liver noses, lips and eye rims. Coat colors may include black, red, red-fawn, liver or variations thereof.
CDA is characterized by loss of hair from dilutely pigmented areas. Coats are normal at birth, and onset of hair loss usually begins between six months and three years of age. Hair loss usually begins along the dorsal midline (middle of the back) and often spares the head, tail and limbs. The pattern seems to vary from breed to breed. It has been suggested(6) that darker colored (steel blue) individuals are less likely to be affected, may be less severely affected or may start to lose hair later in life than lighter colored dogs. This suggests that the severity of the disease may be related to the amount of dilution present. Deeply pigmented or white areas of coat are unaffected. In blue dogs with tan points (Yorkies and Dobermans) the tan areas retain a normal appearance. In piebald (white spotted) individuals, the white areas are unaffected by the hair loss. The hair loss may be total or partial and any remaining hairs are usually sparse, rough and easily broken or removed. The skin in the affected areas is usually scaly and may occasionally develop bacterial infections. Pruritus (itching) is usually absent, unless a bacterial infection has set in.
Diagnosis of CDA requires first ruling out other causes of hair loss. Diagnostic tests should include fungal cultures, skin scrapings to check for parasitic mites, etc. CDA often closely resembles endocrine (hormone related) hair loss and the dog should be carefully examined for any other abnormalities, and tested for normal thyroid function. Presence of dilute pigment and a characteristic course of disease also aid in making the diagnosis. Microscopic examination of hairs and\or skin biopsies can be used to confirm the diagnosis.
There is no cure for CDA. Treatment is limited to controlling the scaliness and any associated pruritus with various shampoos or topical treatments.
The cause of CDA is not clearly understood. Microscopic examination of hairs of dilute individuals reveals that the pigment (melanin) forms large granules (macromelanosomes) which are rarely found in deeply pigmented hairs. In dilute individuals with normal appearing coats, these macromelanosomes are not grouped or clumped and cause no distortion of the cuticle (outer covering) of the hair. Dogs with CDA have many large groups or clumps of macromelanosomes which tend to distort the cuticle of the hair. It is hypothesized that this distortion of the cuticle causes the hairs to break easily, resulting in the short stubby hairs commonly found in affected individuals. (See Drawing). It is further hypothesized that the rupture of the hair releases byproducts of pigment formation, which are toxic to the hair follicles. Regrowth of broken hairs is reduced because of damage to the follicles caused by these toxins.
Why in some dilute dogs the macromelanosomes are clumped and in others they are not, is an interesting question at this time. The relationship between dilute pigment and hair loss is clear, but why are some dilute individuals unaffected? Weimeraners as a breed are dd, all individuals are dilute, yet the disease is unreported in this breed. In Dobermans, the dilute individuals comprise only 8-9% of the breed, yet 50-80%6 of the dilute dogs have CDA. In Italian Greyhounds, many individuals are dilutes, yet the IGCA health survey reported only 71 affected individuals among the approximately 2200 dogs included in the survey.(7) If half the dogs included in the survey were dilutes, the incidence of CDA in IG's would be around 7% of the dilute population, as opposed to the 50-80% affected dilute Dobermans.
A third allele (dl) which is associated with CDA has been proposed.6 While this is a long way from being proven, it could help explain why some dilute animals are unaffected. Dogs with a genotype dd would be normal coated dilutes, ddl would be intermediates (mildly affected?) and dldl would be CDA affected. A genotype of Ddl should represent deeply pigmented dogs which were carriers of CDA.
Italian Greyhounds would be an interesting breed to study regarding the heredity of this condition. IG's have a relatively high incidence of dilutes, and a relatively low incidence of CDA affected dogs.
Just so you are aware of it. I would still take my chances getting one of those pups. Could be mange, or hotspots? Let us know how it turns out.
Color Dilution Alopecia
By Teri Dickinson, DVM
Alopecia (hair loss) related to dilute coat color is a recognized condition in dogs. The currently accepted medical terminology for this condition is Color Dilution Alopecia (CDA). The condition may affect any dilutely pigmented dog, regardless of coat color. This condition was previously known as Blue Balding Syndrome, Blue Doberman Syndrome, Color Mutant Alopecia, Congenital Alopecia, etc. The term Color Mutant Alopecia arose because dilutes were at one time mutations from the deep pigment occurring in wild canines. Dilutes are now a regularly occurring form of pigmentation in many breeds and have been for hundreds of years. The term mutation is therefore not applicable to dilute individuals. References to Doberman Pinschers or blue hair coats arose because the condition is common in blue individuals of this breed, but it is not limited to either blue dogs or Dobermans. The term congenital means present at birth, but CDA affected dogs are born with normal hair coats.
The dilute (also known as Maltese) gene also appears in both mice and cats, and interestingly enough, is not associated with any abnormal coat conditions in those species.(1) Color Dilution Alopecia (CDA) has been recognized in dilute individuals of many breeds of dogs including Chow Chows, Dachshunds, Doberman Pinschers, Great Danes, Irish Setters, Italian Greyhounds, Standard Poodles, Salukis, Whippets, and Yorkshire Terriers.((2),(3),(4),(5)) Dilute individuals carry a recessive genotype of dd and are characterized by blue, bluish-grey, lavender or flesh-colored noses, lips and eye rims. The coat colors may include blue, fawn, blue-fawn, bronze, taupe or some variation of these. These dogs are usually easily distinguished from their deeply (non-dilute) pigmented counterparts. Deeply pigmented individuals carry a dominant genotype of Dd or DD and have black or liver noses, lips and eye rims. Coat colors may include black, red, red-fawn, liver or variations thereof.
CDA is characterized by loss of hair from dilutely pigmented areas. Coats are normal at birth, and onset of hair loss usually begins between six months and three years of age. Hair loss usually begins along the dorsal midline (middle of the back) and often spares the head, tail and limbs. The pattern seems to vary from breed to breed. It has been suggested(6) that darker colored (steel blue) individuals are less likely to be affected, may be less severely affected or may start to lose hair later in life than lighter colored dogs. This suggests that the severity of the disease may be related to the amount of dilution present. Deeply pigmented or white areas of coat are unaffected. In blue dogs with tan points (Yorkies and Dobermans) the tan areas retain a normal appearance. In piebald (white spotted) individuals, the white areas are unaffected by the hair loss. The hair loss may be total or partial and any remaining hairs are usually sparse, rough and easily broken or removed. The skin in the affected areas is usually scaly and may occasionally develop bacterial infections. Pruritus (itching) is usually absent, unless a bacterial infection has set in.
Diagnosis of CDA requires first ruling out other causes of hair loss. Diagnostic tests should include fungal cultures, skin scrapings to check for parasitic mites, etc. CDA often closely resembles endocrine (hormone related) hair loss and the dog should be carefully examined for any other abnormalities, and tested for normal thyroid function. Presence of dilute pigment and a characteristic course of disease also aid in making the diagnosis. Microscopic examination of hairs and\or skin biopsies can be used to confirm the diagnosis.
There is no cure for CDA. Treatment is limited to controlling the scaliness and any associated pruritus with various shampoos or topical treatments.
The cause of CDA is not clearly understood. Microscopic examination of hairs of dilute individuals reveals that the pigment (melanin) forms large granules (macromelanosomes) which are rarely found in deeply pigmented hairs. In dilute individuals with normal appearing coats, these macromelanosomes are not grouped or clumped and cause no distortion of the cuticle (outer covering) of the hair. Dogs with CDA have many large groups or clumps of macromelanosomes which tend to distort the cuticle of the hair. It is hypothesized that this distortion of the cuticle causes the hairs to break easily, resulting in the short stubby hairs commonly found in affected individuals. (See Drawing). It is further hypothesized that the rupture of the hair releases byproducts of pigment formation, which are toxic to the hair follicles. Regrowth of broken hairs is reduced because of damage to the follicles caused by these toxins.
Why in some dilute dogs the macromelanosomes are clumped and in others they are not, is an interesting question at this time. The relationship between dilute pigment and hair loss is clear, but why are some dilute individuals unaffected? Weimeraners as a breed are dd, all individuals are dilute, yet the disease is unreported in this breed. In Dobermans, the dilute individuals comprise only 8-9% of the breed, yet 50-80%6 of the dilute dogs have CDA. In Italian Greyhounds, many individuals are dilutes, yet the IGCA health survey reported only 71 affected individuals among the approximately 2200 dogs included in the survey.(7) If half the dogs included in the survey were dilutes, the incidence of CDA in IG's would be around 7% of the dilute population, as opposed to the 50-80% affected dilute Dobermans.
A third allele (dl) which is associated with CDA has been proposed.6 While this is a long way from being proven, it could help explain why some dilute animals are unaffected. Dogs with a genotype dd would be normal coated dilutes, ddl would be intermediates (mildly affected?) and dldl would be CDA affected. A genotype of Ddl should represent deeply pigmented dogs which were carriers of CDA.
Italian Greyhounds would be an interesting breed to study regarding the heredity of this condition. IG's have a relatively high incidence of dilutes, and a relatively low incidence of CDA affected dogs.
Just so you are aware of it. I would still take my chances getting one of those pups. Could be mange, or hotspots? Let us know how it turns out.
Ridge Runner Beagles
http://www.geocities.com/samlyn0001
http://www.geocities.com/samlyn0001
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fair chase
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Chuck Terry
- Posts: 621
- Joined: Sat Jul 27, 2002 9:26 pm
I vote fungus! Here are some things that should help: Hot spot spray (look for tanic acid early in the ingredients), Sardex II (by Happy Jack), Nustock, or Anchor Blue lotion (will need to be applied daily), or mix equal parts sulphur, mineral oil, turpintine, and chlorox. Others claim "rose dust" is great but I have never tried that. Good luck!
UPDATE:
I've been using a mix of Peroxide one day and Blue Kote the next for about a week and a half. The spots seem to not be scabbing up, hair in regrowing, and she is looking much better (other than having a 1/2 purple dog). I'm going to have to stop running her for a week or so because the sores seem to be getting infection after running but she is doing much better.
So.....it must have been some sort of fungus that she picked up from the cuts on her ears and muzzle the one time she was out running.
I've been using a mix of Peroxide one day and Blue Kote the next for about a week and a half. The spots seem to not be scabbing up, hair in regrowing, and she is looking much better (other than having a 1/2 purple dog). I'm going to have to stop running her for a week or so because the sores seem to be getting infection after running but she is doing much better.
So.....it must have been some sort of fungus that she picked up from the cuts on her ears and muzzle the one time she was out running.